HIV I: Molecular Biology and Pathogenesis: Clinical Applications: 49 (Advances in Pharmacology)

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HIV I: Molecular Biology and Pathogenesis: Clinical Applications: 49 (Advances in Pharmacology)

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Possible viral cause of multiple sclerosis. In: ViralInfections of the Nervous System. Philadelphia : Lippincott-Raven , : — Power , C. Retroviral diseases of the nervous system: pathogenichost response or viral gene-mediated neurovirulence? Trends Neurosci ; 24 : — Dual infection of thecentral nervous system by AIDS viruses with distinct cellulartropisms. Science ; : — Human immunodeficiencyvirus and the brain: investigation of virus load and neuropathologic changes in pre-AIDS subjects.

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Comparisons of HIV-1 viralsequences in brain, choroid plexus and spleen: Potential role of choroid plexus in the pathogenesis of HIV encephalitis. Characterization of chemokine receptor utilization of viruses in the latent reservoir for human immunodeficiency virus type 1.

J Virol ; 74 : — Isolates of humanimmunodeficiency virus type 1 from the brain may constitute a special group of the AIDS virus. Sequence analysis of the V3loop in brain and spleen of patients with HIV encephalitis. Demented andnondemented patients with AIDS differ in brain-derived human immunodeficiency virus type 1 envelope sequences. J Virol ; 68 : — Cerebrospinal fluid betachemokine concentrations in neurocognitively impaired individuals infected with human immunodeficiency virus type 1.


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The role of the blood-brainbarrier in HIV infection of the central nervous system. Adv Neuroimmunol ; 4 : — Relationshipbetween human immunodeficiency virus-associated dementia and viral load in cerebrospinal fluid and brain. Ann Neurol ; 42 : — Cellular localization of human immunodeficiency virus infection within the brains of acquired immune deficiency syndrome patients.

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J Virol ; 70 : — Macrophage-tropic humanimmunodeficiency virus isolates from different patients exhibit unusual V3 envelope sequence homogeneity in comparison with T-cell-tropic isolates: definition of critical amino acids involvedin cell tropism. J Virol ; 66 : — Structure of an HIV gpenvelope glycoprotein in complex with the CD4 receptor and a neutralizing human antibody. Chemokines andreceptors in HIV encephalitis.

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HIV Replication 3D Medical Animation

Identification of humanimmunodeficiency virus envelope gene sequences influencing viral entry into CD4-positive HeLa cells, T-leukemia cells, andmacrophages. Macrophagetropism determinants of human immunodeficiency virus type 1in vivo. Mosaic structure of thehuman immunodeficiency virus type 1 genome infecting lymphoid cells and the brain: evidence for frequent in vivo recombination events in the evolution of regional populations. Neuroimmunomodulation ; 5 : — Expression directed fromHIV long terminal repeats in the central nervous system of transgenic mice.


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Lentiviral infection in thebrain induce matrix metalloproteinase expression: the role of envelope diversity. Neuronal death induced bybrain-derived human immunodeficiency virus type 1 envelope genes differs between demented and nondemented AIDSpatients. J Neurovirology ; 6 : — Apoptosis induced by infection ofprimary brain cultures with diverse human immunodeficiency virus type 1 isolates: evidence for a role of the envelope.

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Neuronal injury due to HIV-1 envelope protein is blocked by anti-gp antibodies but notby anti-CD4 antibodies. Neurology ; 40 : — Neuroscience ; 76 : — The envelope glycoprotein ofhuman immunodeficiency virus type 1 stimulates release of neurotoxins from monocytes. The human immunodeficiencyvirus-1 envelope protein gp binds through its V3 sequence to the glycine site of N-methyl-D-aspartate receptors mediating noradrenaline release in the hippocampus.

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HIV protease inhibitors: a review of molecular selectivity and toxicity

The Tat protein of HIV-1 inducestumor necrosis factor-alpha production. Implications for HIVassociated neurological diseases. Transient exposure to HIV-1 Tatprotein results in cytokine production in macrophages and astrocytes. A hit and run phenomenon. HIV-1 Tat neurotoxicity isprevented by matrix metalloproteinase inhibitors. Ann Neurol ; 49 : — Decay characteristics ofHIVinfected compartments during combination therapy. Cerebrospinal fluid humanimmunodeficiency virus type 1 RNA levels are elevated in neurocognitively impaired individuals with acquired immunodeficiency syndrome.

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